Author

Jeffrey M. Wilson

University of VirginiaORCID 0000-0002-5975-1760Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Immunology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Catherine A. BonhamiD· 2 shared Jeffrey M. SturekiD· 2 shared Judith A. WoodfolkiD· 2 shared Alexandra KadliD· 1 shared Andrew Julio BarrosiD· 1 shared Behnam KeshavarziD· 1 shared Chintan Ramani· 1 shared Claire E. CrossiD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersImmunology

Catherine A. BonhamORCID

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3 papers

Recent·Most cited

ACE-2-like Enzymatic Activity in Anti-SARS-CoV-2 Spike Protein Monoclonal Antibodies

Yufeng Song, Frances Mehl, Tom J. No, Lauren Livingston, Juan Sebastián Quintero, Jun Hayashi, Ginette Serrero, Pamela D. Schoppee, Jeffrey M. Wilson, James E. Crowe, David D. Ho, Michael T. Yin, Joshua Tan, Steven L. Zeichner

This study identifies monoclonal antibodies with ACE2-like catalytic activity that may contribute to post-acute sequelae of COVID-19.

bioRxiv · Apr 23, 2026 · PreprintMicrobiology

IgA antibodies against oxidation-specific epitopes are generated by double negative B cells and are associated with impaired lung function in fibrotic interstitial lung diseases.

Eva Otoupalova, Riley T. Hannan, Pearl Raichura, Noora Batrash, Paul Dell, Mônica Josiane Rodrigues‐Jesus, Eli R. Zunder, Jeffrey M. Wilson, Judith A. Woodfolk, Justin J. Taylor, Shwu‐Fan Ma, Andrew Julio Barros, Yun Michael Shim, Imre Noth, Catherine A. Bonham, , Jeffrey M. Sturek

This study identifies elevated anti-oxidation-specific epitope (OSE) IgA antibodies in fibrotic interstitial lung diseases (fILD) and links them to impaired lung function and a specific B cell subset.

bioRxiv · Dec 2, 2025 · PreprintImmunology

Distinct Type 1 Immune Networks Underlie the Severity of Restrictive Lung Disease after COVID-19

Glenda Canderan, Lyndsey M. Muehling, Alexandra Kadl, Shay Ladd, Catherine A. Bonham, Claire E. Cross, Sierra M. Lima, Xihui Yin, Jeffrey M. Sturek, Jeffrey M. Wilson, Behnam Keshavarz, Kyle B. Enfield, Chintan Ramani, Naomi Bryant, Deborah D. Murphy, , Judith A. Woodfolk

The paper identifies distinct type 1 immune networks associated with varying severities of restrictive lung disease following COVID-19.

Nature Immunology · Mar 26, 2025Peer-reviewedImmunology