Author

Julie Frouard

Gladstone InstitutesORCID 0000-0002-6296-2534Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Assay:CITE-seq· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Nadia R. RoaniD· 2 shared Adam T. GrundhoffiD· 1 shared Atul Janardhan ButteiD· 1 shared Cédric FeschotteiD· 1 shared Christoph Schwarz· 1 shared Douglas V. ArnesoniD· 1 shared Eva TolosaiD· 1 shared Frauke Muecksch· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersImmunology

Nadia R. RoanORCID

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2 papers

Recent·Most cited

ImmunologyNature CommunicationsPeer-reviewedMar 3, 2026

HIV-SEQ REVEALS GLOBAL HOST GENE EXPRESSION DIFFERENCES BETWEEN HIV-TRANSCRIBING CELLS FROM VIREMIC AND SUPPRESSED PEOPLE WITH HIV

Julie Frouard, Sushama Telwatte, Xiaoyu Luo…·Steven A. Yukl

The paper develops HIV-seq to capture HIV transcripts for scRNA-seq, revealing distinct transcriptional features of HIV-transcribing cells in viremic and ART-suppressed HIV patients.

11 citationstop 1%

humanscRNA-seq

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MicrobiologybioRxivPreprintSep 22, 2025

Selective persistence of HIV-1-infected T cell clones can occur through immune reprogramming driven by defective, transcriptionally active proviruses

Martin V. Hamann, Lisa Brauckmann, Christoph Schwarz…·U. Schiefer Lange

This study shows that defective HIV-1 proviruses can reprogram CD4+ T cells by altering BACH2 and STAT5B gene expression, leading to immune evasion and persistence in ART-treated individuals.

DOI 10.1101/2025.09.22.676989

humanbulk RNA-seq

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