Author

Frauke Muecksch

Gladstone InstitutesPrimary ORCID profile (0000-0002-0132-5101 · 4 papers)Unclaimed ORCID profile · follow to get notified of new preprints

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Frauke MueckschiD4 papers · Heidelberg University

Biology Disciplines

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Microbiology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

Adam T. GrundhoffiD· 1 shared Cédric FeschotteiD· 1 shared Christoph Schwarz· 1 shared Eva TolosaiD· 1 shared Julie FrouardiD· 1 shared Lisa Brauckmann· 1 shared Maisha AdibaiD· 1 shared Martin V. Hamann· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperMicrobiology

Adam T. GrundhoffORCID

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1 paper

Recent·Most cited

Selective persistence of HIV-1-infected T cell clones can occur through immune reprogramming driven by defective, transcriptionally active proviruses

Martin V. Hamann, Lisa Brauckmann, Christoph Schwarz, Michael Spohn, Ramon Stoeck, Sabrina M. Leddy, Julie Frouard, Maisha Adiba, Riekje Winzer, Sanamjeet Virdi, Adam T. Grundhoff, Cédric Feschotte, Frauke Muecksch, Nadia R. Roan, Eva Tolosa, U. Schiefer Lange

This study shows that defective HIV-1 proviruses can reprogram CD4+ T cells by altering BACH2 and STAT5B gene expression, leading to immune evasion and persistence in ART-treated individuals.

bioRxiv · Sep 22, 2025 · PreprintMicrobiology