Author

Sebastian Kalamajski

Lund UniversityORCID 0000-0002-6600-9302Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

Hindrik MulderiD· 2 shared Paul W. FranksiD· 2 shared Alham Saadat· 1 shared Anahita Soltanian· 1 shared Anant MamidiiD· 1 shared Anders H. RosengreniD· 1 shared Daniel CoraliD· 1 shared Fiona Louise RobertsiD· 1 shared

Activity by Year

2023-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersCell Biology

Hindrik MulderORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Modelling genetic risk of β-cell dysfunction in human induced pluripotent stem cells from patients carrying the MTNR1B risk variant.

T. Singh, Sebastian Kalamajski, João Gabriel Dantas Cunha, Siarhei Hladkou, Fiona Louise Roberts, Sevda Gheibi, Anahita Soltanian, K. Yektay Farahmand, Ola Ekström, Anant Mamidi, Paul W. Franks, Anders H. Rosengren, Henrik Semb, Hindrik Mulder, Malin Fex

This study uses hiPSC and hESC to model the MTNR1B risk variant's effect on β-cell function, finding increased MTNR1B protein and melatonin sensitivity in G-allele carriers.

Journal of Pineal Research · Sep 1, 2025Peer-reviewedCell Biology

Identification of a weight loss-associated causal eQTL in MTIF3 and the effects of MTIF3 deficiency on human adipocyte function

Mi Ni Huang, Daniel Coral, Hamidreza Ardalani, Peter Spégel, Alham Saadat, Melina Claussnitzer, Hindrik Mulder, Paul W. Franks, Sebastian Kalamajski

The paper identifies a causal eQTL at MTIF3 associated with weight loss and shows MTIF3 deficiency affects adipocyte mitochondrial function and lipid metabolism.

eLife · Mar 6, 2023Peer-reviewedGenetics