Judith Bossen

The paper shows that Drosophila tracheal terminal cells are immune-privileged to maintain Foxo-dependent structural plasticity, as immune activation leads to cell death and structural damage.

This study shows that preconception e-nicotine exposure in Drosophila leads to airway developmental defects and physiological abnormalities that persist across generations.

This study identifies JNK as a key integrator of immune and stress signals in airway progenitors, balancing apoptosis and proliferation to maintain tissue homeostasis during inflammation.

This study shows that JAK/STAT signaling in Drosophila airway epithelia must be balanced to maintain homeostasis and stress adaptation, with both insufficient and excessive activity causing pathology.

The paper shows FoxO factors in Drosophila, mouse, and human airway epithelial cells respond to stressors by nuclear translocation, with FoxO-dependent stress pathways altered in asthma across species.