Author

Gentzon Hall

Duke UniversityORCID 0000-0002-6866-9945Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Biophysics· 1 Genetics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

Christopher John SperatiiD· 2 shared Alekshyander Mishra· 1 shared Benjamin SempowskiiD· 1 shared Daniel Silas· 1 shared Karen L. SoldanoiD· 1 shared Malte C. GatheriD· 1 shared Maria E. KovalikiD· 1 shared Megan Stangl· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersBiophysics

Christopher John SperatiORCID

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2 papers

Recent·Most cited

CRB2 Depletion Induces YAP Signaling and Disrupts Mechanosensing in Podocytes

Yingyu Sun, Nils Michael Kronenberg, Sidharth Kumar Sethi, Surjya Narayan Dash, Maria E. Kovalik, Benjamin Sempowski, Shelby Strickland, Rupesh Raina, Christopher John Sperati, Xuefei Tian, Shuta Ishibe, Gentzon Hall, Malte C. Gather

This study shows CRB2 deficiency in podocytes increases YAP signaling and disrupts mechanosensing, leading to enhanced contractility and altered focal adhesion.

American Journal of Physiology-Renal Physiology · Mar 10, 2025Peer-reviewedBiophysics

A Novel TRPC6 Mutation Causes Autosomal Dominant FSGS

Rohan Bhattacharya, Xingrui Mou, Sophia M. Leeman, Alekshyander Mishra, Titilola D. Kalejaiye, Megan Stangl, Daniel Silas, Karen L. Soldano, Christopher John Sperati, Opeyemi A. Olabisi, Gentzon Hall, Samira Musah

The paper reports a novel TRPC6 mutation causing autosomal dominant FSGS and shows that combined Sildenafil and Losartan therapy improves podocyte dysfunction.

bioRxiv · Feb 11, 2025 · PreprintGenetics