Author

Makoto Kanzaki

Tohoku UniversityORCID 0000-0002-6884-2955Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Neuroscience· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:proteomics· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

Naoki SuzukiiD· 2 shared Ayaka TabuchiiD· 1 shared Chitose SuzukiiD· 1 shared Daisuke HoshinoiD· 1 shared Eiichi N. KodamaiD· 1 shared Eiji ItoiiD· 1 shared Eikan MishimaiD· 1 shared Fukushima Takeru· 1 shared

Activity by Year

2020-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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2 shared papersPhysiology

Naoki SuzukiORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Leucine Aminopeptidase 3 Regulates Skeletal Muscle Mitochondrial Homeostasis with Sex-Dependent Metabolic Consequences

Shion Osana, Reiya Murakami, Ryui Natsuyama, Ayaka Tabuchi, Ryotaro Kano, Kohei Baba, Haopeng Wang, Hiroaki Takada, Naoki Suzuki, Kazutaka Murayama, Makoto Kanzaki, Yasuo Kitajima, Mizuki Sudo, Daisuke Hoshino, Ryoichi Nagatomi, Yutaka Kano

This study identifies LAP3 as a regulator of skeletal muscle mitochondrial homeostasis, showing sex-dependent metabolic consequences in mice.

bioRxiv · Jun 25, 2026 · PreprintPhysiology

ACE2 associates with insulin-responsive GLUT4 dynamics in adipocytes

Fukushima Takeru, Natsuho Moriyama, Haya Sato, Hafumi Nishi, Gwyn W. Gould, Makoto Kanzaki

This study shows ACE2 associates with insulin-responsive GLUT4 vesicles in adipocytes, linking metabolic signaling to ACE2 trafficking and shedding.

bioRxiv · Mar 11, 2026 · PreprintCell Biology

Mitochondrial dysfunction underlying sporadic inclusion body myositis is ameliorated by the mitochondrial homing drug MA-5

Yoshitsugu Oikawa, Rumiko Izumi, Masashi Koide, Yoshihiro Hagiwara, Makoto Kanzaki, Naoki Suzuki, Koichi Kikuchi, Tetsuro Matsuhashi, Yukako Akiyama, Mariko Ichijo, Shun Watanabe, Takafumi Toyohara, Takehiro Suzuki, Eikan Mishima, Yasutoshi Akiyama, , Takaaki Abe

This study identifies mitochondrial dysfunction in sporadic inclusion body myositis and shows that MA-5 improves mitochondrial function and protects myoblasts.

PLoS ONE · Dec 2, 2020Peer-reviewedNeuroscience