Author

Naoki Suzuki

Tohoku UniversityORCID 0000-0001-8880-8554Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 3 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 3 Approach:experimental assay· 3 Approach:statistical model· 3 Assay:bulk RNA-seq· 2 Research Focus:drug response / drug discovery· 2 Organism:human· 2 Organism:mouse· 2 Assay:proteomics· 2

Frequent Co-Authors

Makoto KanzakiiD· 2 shared Masashi AokiiD· 2 shared Ayaka TabuchiiD· 1 shared Chitose SuzukiiD· 1 shared Daijiro Inomata· 1 shared Daisuke HoshinoiD· 1 shared Eiichi N. KodamaiD· 1 shared Eiji ItoiiD· 1 shared

Activity by Year

2020-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersPhysiology

Makoto KanzakiORCID

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4 papers

Recent·Most cited

Leucine Aminopeptidase 3 Regulates Skeletal Muscle Mitochondrial Homeostasis with Sex-Dependent Metabolic Consequences

Shion Osana, Reiya Murakami, Ryui Natsuyama, Ayaka Tabuchi, Ryotaro Kano, Kohei Baba, Haopeng Wang, Hiroaki Takada, Naoki Suzuki, Kazutaka Murayama, Makoto Kanzaki, Yasuo Kitajima, Mizuki Sudo, Daisuke Hoshino, Ryoichi Nagatomi, Yutaka Kano

This study identifies LAP3 as a regulator of skeletal muscle mitochondrial homeostasis, showing sex-dependent metabolic consequences in mice.

bioRxiv · Jun 25, 2026 · PreprintPhysiology

ALS-associated RNA binding proteins converge on UNC13A transcription through regulation of REST

Yasuaki Watanabe, Naoki Suzuki, Tadashi Nakagawa, Masaki Hosogane, Tetsuya Akiyama, Hitoshi Warita, Masashi Aoki, Keiko Nakayama

The paper shows that three ALS-associated RBPs regulate UNC13A transcription by targeting REST, highlighting their role in synaptic integrity and ALS pathogenesis.

bioRxiv · Oct 22, 2024 · PreprintNeuroscience

Harnessing Synaptic Vesicle Release and Recycling Mechanism for Molecule Delivery to Neurons

Karen Kar Lye Yee, Junichi Kumamoto, Daijiro Inomata, Naoki Suzuki, Ryuhei Harada, Norihiro Yumoto

This paper demonstrates a neuron-selective drug delivery system using synaptic vesicle release and recycling mechanism via anti-SYT2 antibodies to deliver therapeutic molecules to motor neurons.

bioRxiv · Sep 11, 2024 · PreprintNeuroscience

Mitochondrial dysfunction underlying sporadic inclusion body myositis is ameliorated by the mitochondrial homing drug MA-5

Yoshitsugu Oikawa, Rumiko Izumi, Masashi Koide, Yoshihiro Hagiwara, Makoto Kanzaki, Naoki Suzuki, Koichi Kikuchi, Tetsuro Matsuhashi, Yukako Akiyama, Mariko Ichijo, Shun Watanabe, Takafumi Toyohara, Takehiro Suzuki, Eikan Mishima, Yasutoshi Akiyama, , Takaaki Abe

This study identifies mitochondrial dysfunction in sporadic inclusion body myositis and shows that MA-5 improves mitochondrial function and protects myoblasts.

PLoS ONE · Dec 2, 2020Peer-reviewedNeuroscience