Author

Yoshihiro Hagiwara

Tohoku UniversityORCID 0000-0002-2992-2178Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

Chitose SuzukiiD· 1 shared Eiichi N. KodamaiD· 1 shared Eiji ItoiiD· 1 shared Eikan MishimaiD· 1 shared Hironori HayashiiD· 1 shared Ken‐ichiro HayashiiD· 1 shared Koichi Kikuchi· 1 shared Makoto KanzakiiD· 1 shared

Activity by Year

2020-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperNeuroscience

Chitose SuzukiORCID

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1 paper

Recent·Most cited

Mitochondrial dysfunction underlying sporadic inclusion body myositis is ameliorated by the mitochondrial homing drug MA-5

Yoshitsugu Oikawa, Rumiko Izumi, Masashi Koide, Yoshihiro Hagiwara, Makoto Kanzaki, Naoki Suzuki, Koichi Kikuchi, Tetsuro Matsuhashi, Yukako Akiyama, Mariko Ichijo, Shun Watanabe, Takafumi Toyohara, Takehiro Suzuki, Eikan Mishima, Yasutoshi Akiyama, , Takaaki Abe

This study identifies mitochondrial dysfunction in sporadic inclusion body myositis and shows that MA-5 improves mitochondrial function and protects myoblasts.

PLoS ONE · Dec 2, 2020Peer-reviewedNeuroscience