Author

Swati Dubey

University of California, Los AngelesORCID 0000-0002-6957-4294Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Swati Dubey1 paper · University of Florida

Biology Disciplines

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Cancer Biology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Organism:bacteria (other)· 1 Assay:bulk RNA-seq· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

Fausto J. RodrígueziD· 2 shared Ajeeth Iyer· 1 shared Albert LaiiD· 1 shared Charles G. EberhartiD· 1 shared Christopher Tse· 1 shared David A. NathansoniD· 1 shared Fabiola Guillen· 1 shared Guanqiao Yu· 1 shared

Activity by Year

2024-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersCancer Biology

Fausto J. RodríguezORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Beyond Cell Cycle Control: CDKN2A Loss Orchestrates NAD⁺ Metabolic Plasticity and NAMPT Inhibitor Sensitivity in Glioblastoma

Swati Dubey, Guanqiao Yu, Ryana Aboul-Hosn, Christopher Tse, David A. Nathanson, Albert Lai, Keith A. Vossel, Fausto J. Rodríguez

The paper shows CDKN2A loss in glioblastoma affects NAD+ metabolism and increases sensitivity to NAMPT inhibitors, beyond its role in cell cycle control.

bioRxiv · Dec 4, 2025 · PreprintCancer Biology

Master control of protein secretion by Mycobacterium tuberculosis

Rashmi Ravindran Nair, Virginia Meikle, Swati Dubey, Mikhail Pavlenok, Michael Niederweis

This study identifies a master control mechanism for protein secretion in M. tuberculosis involving the ESX-4 system and reveals its role in virulence and host cell interaction.

bioRxiv · Mar 13, 2025 · PreprintMicrobiology

High-Throughput Targeted Drug Screening for NF1-associated High-Grade Gliomas with ATRX Deficiency

Swati Dubey, Simran Rai, Fabiola Guillen, Ajeeth Iyer, Su Aung, Ming Yuan, Charles G. Eberhart, Fausto J. Rodríguez

This study identifies K784-6195 as a selective cytotoxic compound for NF1-associated high-grade gliomas with ATRX deficiency, showing reduced sensitivity in wild-type ATRX cells and metabolic pathway disruption.

bioRxiv · Dec 14, 2024 · PreprintCancer Biology

Swati Dubey · henhao