Author

Sandrine Placier

InsermORCID 0000-0002-7075-4655Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Assay:clinical cohort / trial· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

Charles VerneyiD· 1 shared Chloé RafaeliD· 1 shared Christine Walter· 1 shared David LegouisiD· 1 shared Deborah Paolucci· 1 shared Eric FérailleiD· 1 shared Frédéric SanglaiD· 1 shared Gilles CrambertiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperPhysiology

Charles VerneyORCID

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2 papers

Recent·Most cited

Renal adaptation to high salt diet requires tubular Na+ secretion through type A intercalated cells

Chloé Rafael, Luciana Morla, Justine Billiet, Lydie Cheval, Samia Lasaad, Sandrine Placier, Christine Walter, Nicolas Picard, Gilles Crambert

The paper identifies type A intercalated cells as critical for Na+ secretion in response to high salt diets and shows their absence leads to a Bartter-like phenotype.

bioRxiv · Feb 4, 2026 · PreprintPhysiology

The preferential injury of outer renal medulla after ischemia-reperfusion relies on high oxidative metabolism

Grégoire Arnoux, Justine Serre, Thomas Verissimo, Matthieu Tihy, Sandrine Placier, Charles Verney, Frédéric Sangla, Deborah Paolucci, Marylise Fernandez, Sophie de Seigneux, Sebastian Douglas Sgardello, Maarten Naesens, Juliette Hadchouel, Eric Féraille, Stellor Nlandu Khodo, , David Legouis

This study identifies the outer stripe of the outer medulla as the most metabolically active and injured region in the kidney during ischemia-reperfusion injury, showing propofol exacerbates this damage through mitochondrial and fatty acid oxidation impairment.

bioRxiv · Sep 12, 2024 · PreprintPhysiology