Shashi Kumar Suman

This study uses advanced microscopy to show that myosin II minifilaments persist and recycle in C. elegans embryos, contributing to cortical contractility through stable, static interactions with actin.

The paper identifies lin-15A as a Licenser that facilitates transdifferentiation by antagonizing identity safeguarding mechanisms in C. elegans.

The paper investigates the kinetic delays in the RhoA signaling cascade during actomyosin contractility in C. elegans embryos and shows they actively control force deployment.

This paper develops genetically encoded fluorescent reporters to quantify actin filament organization in living cells and tissues using fluorescence polarization microscopy.

This study shows that non-muscle myosins NMY-1 and NMY-2 are required for late C. elegans embryonic elongation through redundant roles in epidermal remodeling.

The paper investigates a natural transdifferentiation event in C. elegans, showing that Wnt signaling and reprogramming factors work together to erase rectal identity and establish neuronal identity during cell division.

This study maps the dynamic localization of brush border components in C. elegans using high-resolution imaging and genome editing, showing dynamic enrichment during microvilli formation and stability afterward.

The paper uses CRISPR to study the role of VAB-10's plakin domain in mechanotransduction during C. elegans morphogenesis, showing its importance in hemidesmosome function and embryonic elongation.