Author

Tingting Wang

Flinders UniversityORCID 0000-0002-7739-4787Unclaimed ORCID profile · follow to get notified of new preprints

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Tingting Wang2 papers · Shanghai Medical College of Fudan University

Biology Disciplines

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Biochemistry· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Janni PeterseniD· 2 shared Abner Gonzalez Castro· 1 shared Anthony R. MeansiD· 1 shared Christopher R. HorneiD· 1 shared David Ernest JamesiD· 1 shared Esben SøndergaardiD· 1 shared Gerard ManningiD· 1 shared Hannah HuckstepiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersPhysiology

Janni PetersenORCID

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2 papers

Recent·Most cited

Personalized phosphoproteomics establish mTORC1 as a regulator of exercise-induced insulin sensitization in human skeletal muscle

Magnus R. Leandersson, Hannah Huckstep, Johan Onslev, Nicolai S. Henriksen, Jonas M. Kristensen, M. N. Zankel, R. Elmquist, Joachim Thorup Edberg Madsen, Jakob Solgaard Jensen, Jesper B. Birk, Kim Anker Sjøberg, Tingting Wang, Janni Petersen, Jerry Richard Greenfield, David Ernest James, , Jørgen F. P. Wojtaszewski

The paper uses personalized phosphoproteomics to show that mTORC1 regulates exercise-induced insulin sensitization in human skeletal muscle through a feedback pathway involving MKNK2 and eIF4G1.

bioRxiv · Sep 2, 2026 · PreprintPhysiology

Unconventional binding of Calmodulin to CHK2 kinase inhibits catalytic activity

Christopher R. Horne, Tingting Wang, Samuel N. Young, Toby A. Dite, Hunter G. Nyvall, Sushant Suresh, Katherine A. Davies, Abner Gonzalez Castro, Vineet Vaibhav, Lucy J. Mather, Laura F. Dagley, Matthew J. Belousoff, Gerard Manning, Anthony R. Means, John E. Burke, , James M. Murphy

The paper identifies an unconventional mechanism where Ca2+-CaM binds CHK2 kinase to inhibit its activity, contrasting with its typical role in promoting kinase activity.

Biochemical Journal · Sep 26, 2025Peer-reviewedBiochemistry