Author

Poonam Roshan

Saint Louis UniversityORCID 0000-0002-8104-4771Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Research Focus:perturbation response prediction· 1

Frequent Co-Authors

Riley Luebbers· 2 shared Sofia S. OrigantiiD· 2 shared Abhinav VayyetiiD· 1 shared Aidan Antony· 1 shared Aparna Biswas· 1 shared Ayush MistryiD· 1 shared Edwin AntonyiD· 1 shared Frank A. TedeschiiD· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersBiochemistry

Riley Luebbers

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

BiochemistryNature CommunicationsPeer-reviewedDec 9, 2025

Mechanism of RPA phosphocode priming and tuning by Cdk1/Wee1 signaling circuit

Poonam Roshan, Vikas Kaushik, Ayush Mistry…·Sofia S. Origanti

This study identifies a feedback mechanism involving Cdk1/Wee1 signaling that primes and tunes RPA phosphorylation, crucial for cell cycle progression and DNA damage response.

3 citations

experimental assaychromatin & epigenetic regulation

𝕏 PostBluesky

Molecular BiologybioRxivPreprintJun 24, 2024

Modulation of ribosomal subunit associations by eIF6 is critical for mitotic exit and cancer progression.

Poonam Roshan, Aparna Biswas, Stella Anagnos…·Sofia S. Origanti

This study shows that eIF6 modulates ribosomal subunit associations, affecting mitotic exit and cancer progression by altering translation efficiency of mitotic factors.

DOI 10.1101/2024.06.24.600220

proteomicsstatistical model

𝕏 PostBluesky