Author

Alice Bertero

University of PisaORCID 0000-0002-8147-4341Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:mouse· 4 Assay:imaging· 3 Research Focus:cell-cell communication· 2 Research Focus:differential expression· 2 Research Focus:database / resource· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1

Frequent Co-Authors

Alberto GalbuseraiD· 3 shared Alessandro GozziiD· 3 shared Massimo PasqualettiiD· 3 shared Noemi BarsottiiD· 3 shared Adam Liska· 2 shared Mara SabbioniiD· 2 shared Marco PaganiiD· 2 shared María Luisa ScattoniiD· 2 shared

Activity by Year

2017-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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3 shared papersNeuroscience

Alberto GalbuseraORCID

Mutual co-authors in cluster

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4 papers

Recent·Most cited

Midbrain dopamine neuronal activity modulates splenic immunity through a brain-to-body circuit

Adithya Gopinath, Alfonso junior Apicella, Alice Bertero, Leah T. Phan, Emily Miller, Jordan Seibold, Shannon C. Wall, Jordan Follett, Aidan R. Smith, Elijah Freedman, Lauryn Hornstein, Logan Wood, Hannah Ciupe, Diya Behal, Arianna Grillo, , Habibeh Khoshbouei

This study shows midbrain dopamine neurons modulate splenic immunity via a brain-to-body circuit involving the DVC and celiac ganglion, reducing spleen weight and naive CD4+ T-cell populations.

bioRxiv · Dec 5, 2024 · PreprintNeuroscience

A cross-species link between mTOR-related synaptic pathology and functional hyperconnectivityin autism

Marco Pagani, Noemi Barsotti, Alice Bertero, Stavros Trakoshis, Laura Ulysse, Andrea Locarno, Ieva Misevičiūtė, Alessia De Felice, Carola Canella, Kaustubh Supekar, Alberto Galbusera, Vinod Menon, Raffaella Tonini, Gustavo Deco, Michael Vincent Lombardo, , Alessandro Gozzi

The paper links mTOR-related synaptic pathology to functional hyperconnectivity in autism using mouse models and human fMRI data.

Nature Communications · Oct 19, 2021Peer-reviewedNeuroscience

Deletion of autism risk gene Shank3 disrupts prefrontal connectivity

Marco Pagani, Alice Bertero, Adam Liska, Alberto Galbusera, Mara Sabbioni, Noemi Barsotti, Nigel Colenbier, Daniele Marinazzo, María Luisa Scattoni, Massimo Pasqualetti, Alessandro Gozzi

This study shows that deleting the autism risk gene Shank3 in mice disrupts prefrontal connectivity and is linked to social communication deficits.

Journal of Neuroscience · May 6, 2019Peer-reviewedNeuroscience

Homozygous loss of autism-risk gene CNTNAP2 results in reduced local and long-range prefrontal functional connectivity

Adam Liska, Alice Bertero, Ryszard S. Gomolka, Mara Sabbioni, Alberto Galbusera, Noemi Barsotti, Stefano Panzeri, María Luisa Scattoni, Massimo Pasqualetti, Alessandro Gozzi

This study shows that homozygous loss of CNTNAP2 in mice reduces prefrontal and midline brain functional connectivity, linking it to autism-related traits.

Cerebral Cortex · Jan 30, 2017Peer-reviewedNeuroscience