Author

Emma F Garland

University of SheffieldORCID 0000-0002-8501-4326Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 2 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:imaging· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

Agnieszka N. UrbanekiD· 2 shared Axel AbeleiniD· 2 shared J. Robin HighleyiD· 2 shared Jan JohanssoniD· 2 shared Suman DeiD· 2 shared Willem Hendrik MolenkampiD· 2 shared Alexander J. DeariD· 1 shared Aurélie Schwartzentruber· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

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Focal author (center)Selected co-authorORCID co-authorUnlinked name

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2 shared papersBiophysics

Agnieszka N. UrbanekORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Two functionally distinct amyloid-β intermediates link aggregation pathways to pathology in Alzheimer's disease

Agnieszka N. Urbanek, Alexander J. Dear, David Rhodes, Emma F Garland, Hollie E. Wareing, Harry J Baker, Willem Hendrik Molenkamp, Ruixin Zhang, Gefei Chen, Axel Abelein, Simon Bell, Juan Alberto Varela, Thomas C. T. Michaels, J. Robin Highley, Georg Meisl, , Suman De

This study identifies two distinct amyloid-β intermediates that drive Alzheimer's pathology and shows their suppression reduces plaque burden and improves cognition in mice.

bioRxiv · Oct 1, 2026 · PreprintBiophysics

Secondary nucleation of α-Synuclein drives Mitochondria dysfunctions and Lewy body formation in Parkinson's Disease

Emily E. Prescott, Jiapeng Wei, Emma F Garland, Agnieszka N. Urbanek, Francesco Capriglia, Willem Hendrik Molenkamp, David P Rhodes, Louise D Heywood, Aurélie Schwartzentruber, Elezabeth Stephen, Rachel M. Hughes, Oliver Bandmann, Mark O. Collins, Sara Snogerup Linse, Axel Abelein, , Suman De

This study identifies secondary nucleation of α-Synuclein as the key mechanism driving mitochondrial dysfunction and Lewy body formation in Parkinson's Disease.

bioRxiv · Sep 17, 2025 · PreprintNeuroscience