Natalia A. Riobo‐Del Galdo

This paper shows that inhibiting the HOTAIR/EZH2 interaction with AQB reduces pro-fibrotic gene expression and tissue remodeling in SSc fibroblasts and skin.

This study identifies AURKA/HDAC6 activation as a driver of primary cilia shortening and fibroblast activation in systemic sclerosis.

This study identifies ciliary program imbalance as a driver of fibroblast-to-myofibroblast reprogramming in systemic sclerosis, suggesting ciliotherapy as a potential treatment.

This study shows UBR5 interacts with the HH pathway to regulate skeletal homeostasis and identifies potential targets for biomedical applications.