Author

Katarzyna Starowicz

University of BirminghamORCID 0000-0002-9144-8630Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Joanna R. MorrisiD· 3 shared Alexander J. GarviniD· 2 shared George E. RonsoniD· 2 shared Ruth M. DenshamiD· 2 shared Alexander J. Lanz· 1 shared Alexandra K. Walker· 1 shared Andrew David BeggsiD· 1 shared Aneika C. LeneyiD· 1 shared

Activity by Year

2018-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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3 shared papersMolecular Biology

Joanna R. MorrisORCID

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Author profile →

3 papers

Recent·Most cited

SUMO4 promotes SUMO deconjugation required for DNA double-strand break repair.

Alexander J. Garvin, Alexander J. Lanz, George E. Ronson, Matthew J.W. Mackintosh, Katarzyna Starowicz, Alexandra K. Walker, Yara Aghabi, Hannah L. Mackay, Ruth M. Densham, Jai S. Bhachoo, Aneika C. Leney, Joanna R. Morris

This study identifies SUMO4 as a critical regulator of DNA double-strand break repair by promoting SUMO deconjugation through SENP1 and preventing excessive SUMOylation.

Molecular Cell · Mar 1, 2025Peer-reviewedMolecular Biology

RAD52 underlies the synthetic-lethal relationship between BRCA1/2 and 53BP1 deficiencies and DNA polymerase theta loss.

Katarzyna Starowicz, George E. Ronson, Elizabeth J. Anthony, Lucy Clarke, Alexander J. Garvin, Andrew David Beggs, Celina Whalley, Matthew Edmonds, James F. J. Beesley, Joanna R. Morris

The paper identifies RAD52 as the mediator of synthetic-lethal interactions between BRCA1/2, 53BP1 deficiencies, and DNA polymerase theta loss in murine cells.

bioRxiv · Mar 20, 2022 · PreprintMolecular Biology

PIN1 isomerisation of BRCA1 promotes replication fork protection.

Manuel Daza-Martin, Mohammed Jamshad, Katarzyna Starowicz, Anoop Singh Chauhan, James F.J. Beesley, Jennifer L. Coles, Grant S. Stewart, Ruth M. Densham, Joanna R. Morris

This study identifies PIN1-mediated BRCA1 isomerisation as a regulator of replication fork protection through enhanced BRCA1-BARD1-RAD51 interaction, distinct from BRCA1's role in homologous recombination.

bioRxiv · Nov 28, 2018 · PreprintCell Biology