Author

Antonio M. Fidaleo

Albany Medical CollegeORCID 0000-0002-9254-2573Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Neuroscience· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Alexander A. MonginiD· 3 shared Marianne Touma BoulosiD· 2 shared Aayan M. AfghaniD· 1 shared Alejandro Pablo AdamiD· 1 shared Alena RudkouskayaiD· 1 shared Alexandra L. SchoberiD· 1 shared Alexandra R. Paul· 1 shared Harmaan S. SidhuiD· 1 shared

Activity by Year

2023-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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3 shared papersPharmacology & Toxicology

Alexander A. MonginORCID

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3 papers

Recent·Most cited

Binding to Albumin and Off-Target Toxicity Confound the Use of LRRC8/VRAC Channel Blockers in Cell Physiology Assays

Marianne Touma Boulos, Aayan M. Afghan, Alena Rudkouskaya, Antonio M. Fidaleo, Harmaan S. Sidhu, Meraj Khan, Alexander A. Mongin

This study shows that VRAC blockers have off-target effects and cytotoxicity masked by serum albumin, suggesting they are not reliable for studying VRAC function.

bioRxiv · Jul 19, 2026 · PreprintPharmacology & Toxicology

LRRC8A-containing anion channels promote glioblastoma proliferation via a WNK1/mTORC2-dependent mechanism

Antonio M. Fidaleo, Martin D. Bach, Shaina Orbeta, Iskandar F. Abdullaev, Nina Martino, Alejandro Pablo Adam, Marianne Touma Boulos, Nickolai O. Dulin, Alexandra R. Paul, Yu-Hung Kuo, Alexander A. Mongin

This study identifies LRRC8A-containing anion channels as promoters of glioblastoma proliferation through a WNK1/mTORC2-dependent mechanism.

The Journal of Physiology · Feb 4, 2026Peer-reviewedCancer Biology

Conditional deletion of LRRC8A in the brain protects against stroke damage independently of effect on swelling-activated glutamate release

Mustafa Balkaya, Preeti Dohare, Sophie Chen, Alexandra L. Schober, Antonio M. Fidaleo, Julia W. Nalwalk, Rajan Sah, Alexander A. Mongin

This study shows that deleting LRRC8A in the brain reduces stroke damage without affecting glutamate release, challenging the idea that VRACs contribute to stroke via glutamate release.

iScience · Apr 14, 2023Peer-reviewedNeuroscience