Author

Jacob P. Matson

University of North Carolina at Chapel HillORCID 0000-0002-9375-1676Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 2

Signature Assays, Organisms & Methods

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Organism:Drosophila· 1 Approach:experimental assay· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

Jeanette Gowen CookiD· 2 shared Adam J. Harvey· 1 shared Alistair T. PagnamentaiD· 1 shared Anja‐Katrin BielinskyiD· 1 shared Brenda R. S. TempleiD· 1 shared Colette B. RogersiD· 1 shared Debashree BasuiD· 1 shared Edward M. Blair· 1 shared

Activity by Year

2018-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersMolecular Biology

Jeanette Gowen CookORCID

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2 papers

Recent·Most cited

Bi-allelic MCM10 mutations cause telomere shortening with immune dysfunction and cardiomyopathy

Ryan M. Baxley, Wendy Leung, Megan Schmit, Jacob P. Matson, Lulu Yin, Marissa K. Oram, Liangjun Wang, John Taylor, Jack Hedberg, Colette B. Rogers, Adam J. Harvey, Debashree Basu, Jenny C. Taylor, Alistair T. Pagnamenta, Helene Marie-Pierre Dreau, , Anja‐Katrin Bielinsky

This study identifies bi-allelic MCM10 mutations causing telomere shortening, immune dysfunction, and cardiomyopathy by impairing DNA replication and telomerase activity.

Nature Communications · Mar 12, 2021Peer-reviewedMolecular Biology

Cdt1 variants reveal unanticipated aspects of interactions with Cyclin/CDK and MCM important for normal genome replication.

Pedro N. Pozo, Jacob P. Matson, Yasemin Cole, Katarzyna M. Kedziora, Gavin D. Grant, Brenda R. S. Temple, Jeanette Gowen Cook

The paper investigates Cdt1 variants associated with Meier-Gorlin syndrome and a Drosophila allele to uncover their effects on MCM loading, Cyclin/CDK interactions, and genome replication.

Molecular Biology of the Cell · Oct 3, 2018Peer-reviewedMolecular Biology