Author

Valeria Ramaglia

University of TorontoORCID 0000-0002-9401-5988Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:cell-cell communication· 1 Approach:deep learning· 1

Frequent Co-Authors

Jennifer L. GommermaniD· 3 shared Alexandros PolyzoisiD· 1 shared Alfonso Mauro Malpede· 1 shared Anna S. Blazier· 1 shared Annie PuiD· 1 shared Daniel S. ReichiD· 1 shared David S Guttman· 1 shared Dennis S. W. LeeiD· 1 shared

Activity by Year

2021-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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3 shared papersImmunology

Jennifer L. GommermanORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

ImmunologybioRxivPreprintJun 9, 2025

Maintenance of chronic neuroinflammation in multiple sclerosis via interferon signaling and CD8 T cell-mediated cytotoxicity

Syed Ali Raza, Yoshimi Enose‐Akahata, Anna S. Blazier…·Daniel S. Reich

This study identifies chronic neuroinflammation in MS through scRNA-seq and proteomics, showing persistent IFN signaling and CD8 T cell cytotoxicity despite B-cell depletion.

1 citation

humanscRNA-seq

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ImmunologybioRxivPreprintMay 27, 2024

Mining for disease-associated microbial metabolites in an age-dependent model of multiple sclerosis

Annie Pu, Naomi M. Fettig, Alexandros Polyzois…·Lisa C. Osborne

The paper identifies a microbial metabolite, indole 3-propionic acid, that can reverse non-remitting EAE in aged mice, suggesting therapeutic potential for progressive MS.

3 citations

mousemetabolomics

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NeuroscienceActa NeuropathologicaPeer-reviewedJun 25, 2021

Complement-associated loss of CA2 inhibitory synapses in the demyelinated hippocampus impairs memory

Valeria Ramaglia, Mohit Dubey, Alfonso Mauro Malpede…·Maarten H. P. Kole

The paper investigates complement system involvement in synapse loss in MS hippocampus, finding C1q deposits linked to inhibitory synapse loss and impaired memory.

62 citationstop 2%

humanmouse

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