Author

Ege A Yalcinbas

Johns Hopkins UniversityORCID 0000-0002-9480-7192Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:differential expression· 3 Assay:bulk RNA-seq· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:drug response / drug discovery· 1 Assay:electronic health records· 1 Approach:experimental assay· 1

Frequent Co-Authors

Kristen R. MaynardiD· 2 shared Leonardo Collado‐TorresiD· 2 shared Nicholas J. EaglesiD· 2 shared Anandita Rajpurohit· 1 shared Atharv ChandraiD· 1 shared Bukola AjanakuiD· 1 shared Christian CazaresiD· 1 shared Christina M. GremeliD· 1 shared

Activity by Year

2021-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersNeuroscience

Kristen R. MaynardORCID

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3 papers

Recent·Most cited

Transcriptional response to chronic long-access fentanyl self-administration in rat habenula and amygdala

Robin Magnard, Daianna González‐Padilla, Ege A Yalcinbas, Emma Chaloux-Pinette, Nicholas J. Eagles, Michael S. Totty, Patricia H. Janak, Leonardo Collado‐Torres, Kristen R. Maynard

This study identifies transcriptomic changes in rat habenula and amygdala after chronic fentanyl self-administration, linking them to human cell types and risk genes for addiction.

Addiction Biology · Jul 1, 2026Peer-reviewedNeuroscience

Transcriptomic analysis of the human habenula in schizophrenia

Ege A Yalcinbas, Bukola Ajanaku, Erik D. Nelson, Renee Garcia-Flores, Nicholas J. Eagles, Kelsey D. Montgomery, Joshua M. Stolz, Joshua H. Wu, Heena R. Divecha, Atharv Chandra, Rahul A. Bharadwaj, Svitlana V. Bach, Anandita Rajpurohit, Ran Tao, Geo M. Pertea, , Kristen R. Maynard

The paper performs transcriptomic analysis of the human habenula and identifies molecular changes associated with schizophrenia risk and illness state.

American Journal of Psychiatry · Nov 1, 2025Peer-reviewedNeuroscience

Mechanism for differential recruitment of orbitostriatal transmission during outcomes and actions in alcohol dependence

Rafael Renteria, Christian Cazares, Emily T. Baltz, Drew C. Schreiner, Ege A Yalcinbas, Thomas Steinkellner, Thomas S. Hnasko, Christina M. Gremel

This study identifies a mechanism by which alcohol dependence disrupts OFC-DS circuit activity through D1R SPN dysfunction and increased eCB signaling, impairing outcome-based decision-making.

eLife · Mar 17, 2021Peer-reviewedNeuroscience