Author

Yu‐Juei Hsu

Tri-Service General HospitalORCID 0000-0002-9696-612XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Organism:mouse· 1 Organism:rat· 1

Frequent Co-Authors

Anish K. AminiD· 1 shared Colin FarquharsoniD· 1 shared Gwo‐Jyh ChangiD· 1 shared Ineke H. N. LuijteniD· 1 shared Kanchan PhadwaliD· 1 shared Katherine Ann StainesiD· 1 shared Katie EmelianovaiD· 1 shared Louise A. StepheniD· 1 shared

Activity by Year

2023-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperGenomics

Anish K. AminORCID

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2 papers

Recent·Most cited

Mitochondrial dysfunction and mitophagy blockade contribute to renal osteodystrophy in chronic kidney disease-mineral bone disorder

Shun‐Neng Hsu, Louise A. Stephen, Kanchan Phadwal, Scott Dillon, Roderick Carter, Nicholas M. Morton, Ineke H. N. Luijten, Katie Emelianova, Anish K. Amin, Vicky Elizabeth MacRae, Tom C. Freeman, Yu‐Juei Hsu, Katherine Ann Staines, Colin Farquharson

This study identifies mitochondrial dysfunction and impaired mitophagy in CKD-MBD as contributors to renal osteodystrophy, showing reversible effects with rapamycin treatment.

Kidney International · Feb 6, 2025Peer-reviewedGenomics

Empagliflozin reduces neointimal formation and vascular smooth muscle cell proliferation via the suppression of PDGF-related signaling

Wei-Jan Chen, Gwo‐Jyh Chang, Yu‐Juei Hsu, Ying‐Hwa Chen

This study shows empagliflozin reduces neointimal formation and VSMC proliferation by suppressing PDGF-related signaling, independent of SGLT2 and glucose metabolism.

bioRxiv · Aug 14, 2023 · PreprintMolecular Biology