Author

Shun‐Neng Hsu

Tri-Service General HospitalORCID 0000-0002-8014-6311Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

Colin FarquharsoniD· 2 shared Katherine Ann StainesiD· 2 shared Louise A. StepheniD· 2 shared Scott DilloniD· 2 shared Vicky Elizabeth MacRaeiD· 2 shared Amanda NovakiD· 1 shared Andrew A. PitsillidesiD· 1 shared Anish K. AminiD· 1 shared

Activity by Year

2022-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

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2 shared papersGenomics

Colin FarquharsonORCID

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2 papers

Recent·Most cited

Mitochondrial dysfunction and mitophagy blockade contribute to renal osteodystrophy in chronic kidney disease-mineral bone disorder

Shun‐Neng Hsu, Louise A. Stephen, Kanchan Phadwal, Scott Dillon, Roderick Carter, Nicholas M. Morton, Ineke H. N. Luijten, Katie Emelianova, Anish K. Amin, Vicky Elizabeth MacRae, Tom C. Freeman, Yu‐Juei Hsu, Katherine Ann Staines, Colin Farquharson

This study identifies mitochondrial dysfunction and impaired mitophagy in CKD-MBD as contributors to renal osteodystrophy, showing reversible effects with rapamycin treatment.

Kidney International · Feb 6, 2025Peer-reviewedGenomics

Increased PHOSPHO1 expression mediates cortical bone mineral density in renal osteodystrophy

Shun‐Neng Hsu, Louise A. Stephen, Scott Dillon, Elspeth Milne, Behzâd Javaheri, Andrew A. Pitsillides, Amanda Novak, José Luís Millán, Vicky Elizabeth MacRae, Katherine Ann Staines, Colin Farquharson

This study shows that increased PHOSPHO1 expression in renal osteodystrophy contributes to increased cortical bone mineral density but decreased trabecular bone density.

Journal of Endocrinology · Jan 1, 2022Peer-reviewedMolecular Biology