Author

Melania González-Torres

Universitat Autònoma de BarcelonaORCID 0000-0002-9755-8160Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 1 Assay:proteomics· 1

Frequent Co-Authors

Albert QuintanaiD· 3 shared Elisenda SanziD· 3 shared Aina Redondo· 1 shared Alejandro GellaiD· 1 shared Andrea UrpiiD· 1 shared Carmé Caelles· 1 shared Irene Fernández-GonzáleziD· 1 shared Joan CompteiD· 1 shared

Activity by Year

2020-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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3 shared papersNeuroscience

Albert QuintanaORCID

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3 papers

Recent·Most cited

NeurosciencebioRxivPreprintNov 11, 2025

Sex-dependent hypothalamic microinflammation and microglial polarization: The role of JNK in high-fat diet-induced insulin resistance

Maria Rodriguez-Garcia, Montserrat Bolaños-Hurtado, Aina Redondo…·Rosalía Rodríguez‐Rodríguez

This study investigates sex-specific hypothalamic microinflammation and microglial polarization in response to high-fat diet, identifying JNK3 as a key regulator of these processes in males.

DOI 10.1101/2025.11.11.687241

mouseexperimental assay

𝕏 PostBluesky

NeuroscienceJournal of Clinical InvestigationPeer-reviewedOct 2, 2025

Dysfunctional LHX6 pallido-subthalamic projections mediate epileptic events in a mouse model of Leigh Syndrome

Laura Sánchez-Benito, Melania González-Torres, Irene Fernández-González…·Albert Quintana

This study identifies dysfunctional pallido-subthalamic projections as a mediator of epilepsy in a mouse model of Leigh Syndrome and suggests STN inhibition as a potential therapeutic target.

DOI 10.1172/jci187571

mouseexperimental assay

𝕏 PostBluesky

NeuroscienceFrontiers in Cell and Developmental BiologyPeer-reviewedJul 28, 2020

Mitochondrial Proteome of Affected Neurons in a Mouse Model of Leigh Syndrome

Alejandro Gella, Patricia Prada-Dacasa, Montserrat Carrascal…·Albert Quintana

The paper uses a novel viral tool to isolate mitochondria from affected neurons in a mouse model of Leigh Syndrome, revealing proteomic and acetylomic changes associated with mitochondrial dysfunction.

16 citations

mouseproteomics

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