Author

Neil D. Perkins

Newcastle UniversityORCID 0000-0002-9756-3656Unclaimed ORCID profile · follow to get notified of new preprints

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Neil D. Perkins1 paper

Biology Disciplines

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Bioinformatics· 1 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:imaging· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Andrew Michael FreyiD· 1 shared Andrew PauliD· 1 shared Christopher J. LordiD· 1 shared Christopher P. A. T. LawsoniD· 1 shared D. WilkinsoniD· 1 shared David A. TurneriD· 1 shared David R. AdamsiD· 1 shared Emily StephensoniD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperCancer Biology

Andrew Michael FreyORCID

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2 papers

Recent·Most cited

Domain Specific AI Segmentation of IMPDH2 Rod/Ring Structures in Mouse Embryonic Stem Cells

Sam T. M. Ball, Meagan J. Hennessy, Yuhan Tan, Kai F. Hoettges, Neil D. Perkins, D. Wilkinson, Michael White, Yalin Zheng, David A. Turner

The paper develops an AI pipeline to automatically segment IMPDH2 rod-ring structures in mouse ESCs, achieving high accuracy and agreement with expert annotations.

BMC Biology · May 11, 2025Peer-reviewedBioinformatics

Targeting non-canonical NF-κB signalling in CYLD cutaneous syndrome by selective inhibition of IκB kinase alpha.

Kirsty Hodgson, Joseph Inns, Gary Reynolds, Emily Stephenson, Andrew Paul, Naomi Sinclair, Giacomo Berretta, Christopher P. A. T. Lawson, Andrew Michael Frey, Iglika G. Ivanova, Éva Ádám, Christopher J. Lord, Simon Cockell, Jonathan M Coxhead, Nikoletta Nagy, , Neil Rajan

This study identifies non-canonical NF-κB signalling as a therapeutic target in CYLD cutaneous syndrome and shows that IKK inhibition reduces tumor viability in patient-derived models.

bioRxiv · Jan 31, 2025 · PreprintCancer Biology