David Bartolomé‐Martín

This study investigates how GRIN2B mutations affect NMDA receptor-BK channel coupling, showing that V618G disrupts complex formation and functional coupling, contributing to EIEE27 pathophysiology.

This study shows that SGK1.1 activates the M-current in neurons and can counteract epilepsy-related mutations in Kv7.2 subunits.

The paper shows that BK channels suppress NMDAR activity in barrel cortex neurons, preventing spike-timing-dependent plasticity.