Author

Teresa Giráldez

Universidad de La LagunaORCID 0000-0002-4096-810XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 5 Assay:electronic health records· 3 Organism:mouse· 3 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Assay:electrophysiology· 1 Organism:human· 1

Frequent Co-Authors

Diego Álvarez de la RosaiD· 4 shared David Bartolomé‐MartíniD· 3 shared Elva Martín-BatistaiD· 3 shared Laura Eva MaglioiD· 3 shared Belinda Rivero-PéreziD· 2 shared Guadalberto HernándeziD· 2 shared Natalia Armas-Capote· 2 shared Ricardo J. GómeziD· 2 shared

Activity by Year

2019-2025 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 28 mutual co-author links

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Focal author (center)Selected co-authorORCID co-authorUnlinked name

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4 shared papersNeuroscience

Diego Álvarez de la RosaORCID

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

GRIN2B disease-associated mutations disrupt the function of BK channels and NMDA receptor signalling nanodomains

Rebeca Martínez Lázaro, Teresa Minguez, Andrea Reyes-Carrión, Ricardo J. Gómez, Diego Álvarez de la Rosa, David Bartolomé‐Martín, Teresa Giráldez

This study investigates how GRIN2B mutations affect NMDA receptor-BK channel coupling, showing that V618G disrupts complex formation and functional coupling, contributing to EIEE27 pathophysiology.

The Journal of General Physiology · Jul 8, 2025Peer-reviewedPhysiology

Activation of SGK1.1 up-regulates the M-current in the presence of epilepsy mutations

Elva Martín-Batista, Rían W. Manville, Belinda Rivero-Pérez, David Bartolomé‐Martín, Diego Álvarez de la Rosa, Geoffrey Winston Abbott, Teresa Giráldez

This study shows that SGK1.1 activates the M-current in neurons and can counteract epilepsy-related mutations in Kv7.2 subunits.

Frontiers in Molecular Neuroscience · Nov 26, 2021Peer-reviewedNeuroscience

NMDA receptor-BK channel coupling regulates synaptic plasticity in the barrel cortex

Ricardo J. Gómez, Laura Eva Maglio, Alberto J. Gonzalez-Hernandez, Belinda Rivero-Pérez, David Bartolomé‐Martín, Teresa Giráldez

The paper shows that BK channels suppress NMDAR activity in barrel cortex neurons, preventing spike-timing-dependent plasticity.

Proceedings of the National Academy of Sciences · Aug 27, 2021Peer-reviewedNeuroscience

Neuronal kinase SGK1.1 protects against brain damage after status epilepticus

Elva Martín-Batista, Laura Eva Maglio, Natalia Armas-Capote, Guadalberto Hernández, Diego Álvarez de la Rosa, Teresa Giráldez

This paper shows that SGK1.1 activation reduces neuronal death and gliosis after status epilepticus, acting through both M-current regulation and anti-apoptotic mechanisms.

Neurobiology of Disease · Feb 24, 2021Peer-reviewedNeuroscience

SGK1.1 activation causes early seizure termination via effects on M-current

Natalia Armas-Capote, Laura Eva Maglio, Leonel F. Pérez-Atencio, Elva Martín-Batista, Antonio Reboreda, Juan Antonio Barios, Guadalberto Hernández, Diego Álvarez de la Rosa, José Antonio Lamas, Luis Carlos Barrio, Teresa Giráldez

This study shows that SGK1.1 activation reduces seizure duration and mortality in a mouse model of status epilepticus by increasing M-current in hippocampal neurons.

Cerebral Cortex · Nov 8, 2019Peer-reviewedNeuroscience