Aize Pellón

This study shows the IL-1 family is essential for mucosal defense against Candida albicans, preventing infection and dissemination, but its absence in neutropenic conditions leads to fatal systemic infection.

This study identifies a metabolic-epigenetic mechanism in oral epithelial cells that enhances protection against fungal infection through proline catabolism and mitochondrial function.

This study shows that Candida albicans promotes melanoma aggressiveness by activating p38-MAPK and HIF-1α pathways and inducing metabolic reprogramming.

The paper identifies metabolic differences between Candida species using genomic, metabolomic, and gene expression data, highlighting unique pathways in the AGAu cluster.