Jian Miao

This study shows that fungal-induced immunosurveillance remodels the oral mucosal barrier via a non-canonical IL-22 receptor complex, enhancing resistance to Candida albicans colonization.

This study shows that the commensal fungus Candida albicans enhances Salmonella Typhimurium virulence by promoting arginine production, which in turn boosts Salmonella's ability to invade host cells and dampens the host's inflammatory response.

This study identifies a metabolic-epigenetic mechanism in oral epithelial cells that enhances protection against fungal infection through proline catabolism and mitochondrial function.

This study identifies sulfated glycosaminoglycans as targets of candidalysin, a virulence factor in C. albicans, and shows that blocking these interactions can protect epithelial cells from fungal damage.