Author

Hannah E. Stutzman

Massachusetts General HospitalORCID 0000-0003-0500-437XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Bioengineering· 1 Genetics· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

Ben Kleinstiver· 1 shared Benjamin P. KleinstiveriD· 1 shared C.K. Fong· 1 shared Casey A. MaguireiD· 1 shared Christian Lacks Lino CardenasiD· 1 shared Christiano R. R. AlvesiD· 1 shared Claire E. ShamberiD· 1 shared David Y. ChungiD· 1 shared

Activity by Year

2023-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperGenetics

Ben Kleinstiver

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2 papers

Recent·Most cited

BioengineeringNature Biomedical EngineeringPeer-reviewedSep 11, 2025

In vivo Treatment of a Severe Vascular Disease via a Bespoke CRISPR-Cas9 Base Editor

Christiano R. R. Alves, Sabyasachi Das, Vijai Krishnan…·Patricia Leonor Musolino

This paper develops a customized CRISPR-Cas9 base editor to correct the ACTA2 R179H mutation in a mouse model of MSMDS, improving survival and rescuing vascular and neurological phenotypes.

13 citations

mousegenetic perturbation / CRISPR

𝕏 PostBluesky

GeneticseLifePeer-reviewedDec 19, 2023

Base editing strategies to convert CAG to CAA diminish the disease-causing mutation in Huntington's disease

Doo Eun Choi, Jun Wan Shin, Sophia Zeng…·Jong‐Min Lee

The paper develops base editing strategies to convert CAG to CAA in Huntington's disease, reducing CAG repeat expansion and showing therapeutic potential.

9 citations

mouseexperimental assay

𝕏 PostBluesky