Taylor W. Schmitz

The paper examines how changes in functional connectivity during rest and task relate to Alzheimer's pathology and episodic memory in older adults.

This study examines hippocampal macrostructure and microstructure changes in Alzheimer's Disease and MCI compared to healthy controls using ADNI data.

This study uses frequency-tagged fMRI to show that visual cortical responses are suppressed during inattention and enhanced during attention, supporting the normalization model of attention.

The paper shows that cholinergic synaptic plasticity, specifically VAChT responses to tau, correlates with cognitive resilience in presymptomatic AD.

This study uses frequency-tagging fMRI to show that directed attention enhances anti-phase BOLD oscillations and suppresses in-phase oscillations in visual cortex, supporting the biased competition model of attention.

This paper develops and validates a dual-frequency tagging fMRI protocol to enable fine-grained spatiotemporal analysis of cortical function, revealing detailed cortical topographies and temporal synchronization.

This study examines gradients of basal forebrain connectivity in the human cortex using multimodal imaging and finds reduced tethering between structural and functional connectivity in specific brain regions.

This study examines how neural activity in the primate lateral prefrontal cortex is organized during different cognitive tasks, finding that task-related activity has a consistent spatial scaffold with task-specific reconfiguration.

This study uses meta-analytic neuroimaging to show acetylcholine modulates midcingulo-insular network activity during attention, enhancing response speed without sacrificing accuracy.

Neurotransmitter receptor/transporter PET maps (19 targets, 1239 healthy participants) align with connectomes, explain MEG power, and track cognitive gradients and cortical thinning in 13 disorders.

The paper demonstrates that the right anterior dorsolateral and ventrolateral prefrontal cortex dynamically shift inhibitory connectivity to suppress both actions and thoughts, supporting a domain-general inhibitory control mechanism.

This study shows that basal forebrain volume predicts cortical degeneration in Alzheimer's disease, challenging the traditional view of pathogenesis.