Author

Matthew R. Aronoff

University of New MexicoORCID 0000-0003-0838-7345Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1

Frequent Co-Authors

Amro A. HussieniD· 1 shared Archana G. MohananiD· 1 shared Barbara P. Niederöst· 1 shared Bart GhesquièreiD· 1 shared Diane E. TayloriD· 1 shared Didier SurdeziD· 1 shared Dirk Sw ElewautiD· 1 shared Evi MasscheleiniD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

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10 top collaborators · 45 mutual co-author links

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1 shared paperCell Biology

Amro A. HussienORCID

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1 paper

Recent·Most cited

Cell BiologyScience Translational MedicinePeer-reviewedJan 7, 2026

✦ In-depth digest

HIF1α gates tendon response to overload and drives tendinopathy independently of vascular recruitment

Greta Moschini, Archana G. Mohanan, Izabella S. Niewczas…·Jess Gerrit Snedeker

Mechanical overload activates hypoxia-inducible factor 1-alpha (HIF1a) in tenocytes to drive tendinopathy independently of VEGFA-mediated vascular ingrowth, and tenocyte Hif1a deletion prevents overload-induced tendon degeneration in mice.

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humanmouse

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