Author

Diane E. Taylor

Babraham InstituteORCID 0009-0002-4940-9352Unclaimed ORCID profile · follow to get notified of new preprints

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1

Frequent Co-Authors

Amro A. HussieniD· 1 shared Archana G. MohananiD· 1 shared Barbara P. Niederöst· 1 shared Bart GhesquièreiD· 1 shared Didier SurdeziD· 1 shared Dirk Sw ElewautiD· 1 shared Evi MasscheleiniD· 1 shared Fabian S. PassiniiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCell Biology

Amro A. HussienORCID

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1 paper

Recent·Most cited

HIF1α gates tendon response to overload and drives tendinopathy independently of vascular recruitment

Greta Moschini, Archana G. Mohanan, Izabella S. Niewczas, Diane E. Taylor, Patrick Klaus Jaeger, Guillermo Turiel, Amro A. Hussien, Stefania L. Wunderli, Olivia Baumberger, Maja Wolleb, Florence Marti, Barbara P. Niederöst, Maja Bollhalder, Raphaela Ardicoglu, Nathalie Tisch, , Jess Gerrit Snedeker

Mechanical overload activates hypoxia-inducible factor 1-alpha (HIF1a) in tenocytes to drive tendinopathy independently of VEGFA-mediated vascular ingrowth, and tenocyte Hif1a deletion prevents overload-induced tendon degeneration in mice.

Science Translational Medicine · Jan 7, 2026Peer-reviewedCell Biology✦ In-depth digest