Author

Casey D. Gailey

University of VirginiaORCID 0000-0003-1493-6902Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:C. elegans· 1 Organism:mouse· 1

Frequent Co-Authors

Andrea Cuentas-CondoriiD· 1 shared Ben MulcahyiD· 1 shared David L. BrautiganiD· 1 shared David M. MilleriD· 1 shared Eric J. Wang· 1 shared Hongkyun KimiD· 1 shared Janet E. RichmondiD· 1 shared John A. Tipps· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperNeuroscience

Andrea Cuentas-CondoriORCID

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2 papers

Recent·Most cited

NeuroscienceCell ReportsPeer-reviewedOct 30, 2023

The Epithelial Na+ Channel UNC-8 promotes an endocytic mechanism that recycles presynaptic components from old to new boutons in remodeling neurons

Andrea Cuentas-Condori, Siqi Chen, Mia Krout…·David M. Miller

The paper shows that UNC-8, an ENaC protein, promotes Ca2+-dependent endocytosis to dismantle and recycle presynaptic components during neuronal remodeling in C. elegans.

12 citations

C. elegansexperimental assay

𝕏 PostBluesky

Developmental BiologyDevelopmental DynamicsPeer-reviewedSep 16, 2020

Phosphosite T674A mutation in kinesin family member 3A fails to reproduce tissue and ciliary defects characteristic of CILK1 loss of function

Casey D. Gailey, Eric J. Wang, Li Jin…·Zheng Fu

The paper shows that mutating KIF3A's phosphorylation site T674 to Ala in mice does not reproduce the severe ciliopathy defects seen with CILK1 loss of function.

16 citations

mouseexperimental assay

𝕏 PostBluesky