Author

Jack C. Henry

Queen Mary University of LondonORCID 0000-0003-1521-5146Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Angus J.M. CameroniD· 2 shared Joanne Sewell· 2 shared Neil Q. McDonaldiD· 2 shared Adam M. PassmaniD· 1 shared Alejandro Suárez‐BonnetiD· 1 shared Ashley CampbelliD· 1 shared Francesca HoughtoniD· 1 shared Ivan Quétier· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersCell Biology

Angus J.M. CameronORCID

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2 papers

Recent·Most cited

Cell BiologybioRxivPreprintJun 28, 2026

PKN2 regulates cell-junctions to limit colitis

Jack C. Henry, Ashley Campbell, Prerana Huddar…·Angus J.M. Cameron

This study identifies PKN2 as a regulator of tight junctions that limits colitis and colorectal cancer by maintaining intestinal barrier integrity during inflammation.

DOI 10.64898/2025.12.15.694339

mouseexperimental assay

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Cancer BiologyScience SignalingPeer-reviewedNov 4, 2025

The penetrant chordoid glioma PRKCA mutation is an oncogenic gain-of-function kinase inactivation eliciting early onset chondrosarcoma in mice.

Véronique Calleja, Jack C. Henry, Mathias Cobbaut…·Peter J. Parker

The PRKCA D463H mutation, an inactivating kinase mutation, acts as a gain-of-function oncogenic driver in mice, promoting chondrosarcoma through non-catalytic mechanisms.

1 citation

mouseexperimental assay

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