Author

Luisa María Nieto Ramírez

Colorado State UniversityORCID 0000-0003-1566-5876Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Organism:bacteria (other)· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

David Michael LewinsohniD· 2 shared Karen M. DobosiD· 2 shared William H. HildebrandiD· 2 shared Aneta H. WorleyiD· 1 shared Carolina MehaffyiD· 1 shared Chance LemoniD· 1 shared Corinna A. KulickeiD· 1 shared Deborah A. LewinsohniD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersImmunology

David Michael LewinsohnORCID

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2 papers

Recent·Most cited

ImmunologyJournal of Biological ChemistryPeer-reviewedFeb 26, 2026

Mutations outside the MR1 antigen binding groove differentially inhibit presentation of exogenous antigens

Corinna A. Kulicke, Chance Lemon, Jason R. Krawic…·David Michael Lewinsohn

This study shows that mutations in MR1 outside the antigen binding groove differentially affect exogenous antigen presentation by altering B2M association.

4 citationstop 3%

humanproteomics

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ImmunologyPLoS PathogensPeer-reviewedJul 1, 2025

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Disruption of riboflavin biosynthesis in mycobacteria establishes 5-amino-6-D-ribitylaminouracil (5-A-RU) as key precursor of MAIT cell agonists

Melissa Dalcina Chengalroyen, Nurudeen Oketade, Aneta H. Worley…·David Michael Lewinsohn

Deleting early riboflavin genes (ribA2 in both species, ribG in M. smegmatis) sharply reduced MAIT cell activation, while ribH deletion raised recognition of M. tuberculosis only.

19 citationstop 1%

bacteria (other)human

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