Author

Aneta H. Worley

Oregon Health & Science UniversityORCID 0009-0009-9772-8658Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:proteomics· 2 Organism:bacteria (other)· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Organism:mouse· 1

Frequent Co-Authors

David Michael LewinsohniD· 2 shared Allison E. TammeniD· 1 shared Carolina MehaffyiD· 1 shared Deborah A. LewinsohniD· 1 shared Digby Francis WarneriD· 1 shared Elham KaramooziD· 1 shared Erin J. AdamsiD· 1 shared Gwendolyn M. SwarbrickiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersImmunology

David Michael LewinsohnORCID

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2 papers

Recent·Most cited

ImmunologyScientific ReportsPeer-reviewedMay 29, 2026

Sorting endosomes play key roles in presentation of Mycobacterium tuberculosis-derived ligands to MAIT cells

Allison E. Tammen, Jessie C. Peterson, Aneta H. Worley…·Elham Karamooz

The paper identifies sorting endosomes as critical for MR1 antigen presentation of Mycobacterium tuberculosis-derived ligands to MAIT cells, highlighting the roles of Syntaxin 12 and 16 in this process.

DOI 10.1038/s41598-026-50843-1

humanproteomics

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ImmunologyPLoS PathogensPeer-reviewedJul 1, 2025

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Disruption of riboflavin biosynthesis in mycobacteria establishes 5-amino-6-D-ribitylaminouracil (5-A-RU) as key precursor of MAIT cell agonists

Melissa Dalcina Chengalroyen, Nurudeen Oketade, Aneta H. Worley…·David Michael Lewinsohn

Deleting early riboflavin genes (ribA2 in both species, ribG in M. smegmatis) sharply reduced MAIT cell activation, while ribH deletion raised recognition of M. tuberculosis only.

19 citationstop 1%

bacteria (other)human

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