Andrés M. Baraibar

This study shows that astrocyte CB1R signaling promotes inflammation in MS by disrupting the blood-brain barrier and reducing BBB function.

This study shows that CB1 receptors in neurons and astrocytes differentially affect spatial navigation and memory in male and female mice.

This study shows that human dental pulp stem cells can be differentiated into neuron-like cells with electrophysiological activity, highlighting the importance of serum-free culture protocols.

The paper identifies a spatial threshold for astrocyte calcium signal propagation in mice, which determines gliotransmitter release and synaptic modulation.

This study identifies aberrant astrocytic calcium signaling in MS models as a contributor to synaptic plasticity impairment and gray matter pathology.

This study shows aluminium inhibits calcium, sodium, and potassium currents in bovine chromaffin cells, reducing neurotransmitter release and synaptic transmission.

The paper reveals column- and layer-specific astrocyte-mediated non-synaptic communication between cortical neurons through CB1R activation and ATP/adenosine release.