Kang Ho Kim

The paper identifies conserved molecular responses to cardiometabolic stress across different disease models and finds PAI-1 as a key mediator of cardiac remodeling.

This study identifies PPARα as a regulator of liver proteostasis, showing it suppresses protein synthesis and secretion while enhancing proteasome activity and degradation.

This study shows that deleting Ubc9 in adipocytes causes lipoatrophy and metabolic issues in mice by disrupting adipose tissue expansion and function.