Author

Ilaria Vanni

Istituto Superiore di SanitàORCID 0000-0003-2617-4263Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:proteomics· 2 Organism:C. elegans· 1 Approach:comparative / phylogenetic analysis· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Romolo NonnoiD· 2 shared Abdelkader AmaraiD· 1 shared Alessandra LudovisiiD· 1 shared Andreu Matamoros‐AnglesiD· 1 shared Atef MalekiD· 1 shared Barbara ChiappiniiD· 1 shared Behnam MohammadiiD· 1 shared Berta PuigiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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2 shared papersMicrobiology

Romolo NonnoORCID

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3 papers

Recent·Most cited

MicrobiologyFrontiers in Cellular and Infection MicrobiologyPeer-reviewedApr 7, 2026

Exploring the Repertoire of Rhomboid Proteases in Cryptosporidium parvum Parasite: Phylogenesis, Structural motifs and Cellular Localization in Sporozoite Cells

Ilaria Vanni, Elisabetta Pizzi, Lucia Bertuccini…·Fabio Tosini

This study identifies and characterizes three rhomboid proteases in Cryptosporidium parvum, analyzing their phylogenetic origin, structural features, and subcellular localization in sporozoites.

1 citation

C. elegansproteomics

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MicrobiologybioRxivPreprintSep 17, 2025

First identification of camel prion disease in Tataouine, Tunisia: an emerging animal prion disease in North Africa

Abdelkader Amara, Michele Angelo Di Bari, Kéfia Elmehatli…·Laura Pirisinu

The paper reports six cases of camel prion disease in Tunisia, highlighting its presence in North Africa and suggesting possible contagious transmission mechanisms.

1 citation

multiple speciesexperimental assay

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NeuroscienceActa NeuropathologicaPeer-reviewedJul 9, 2024

Cleavage site-directed antibodies reveal the prion protein in humans is shed by ADAM10 at Y226 and associates with misfolded protein deposits in neurodegenerative diseases

Feizhi Song, Valerija Kovač, Behnam Mohammadi…·Hermann C. Altmeppen

This study identifies ADAM10-mediated shedding of human PrP at Y226 and shows sPrP associates with misfolded protein aggregates in neurodegenerative diseases.

9 citations

humanmouse

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