Author

Zuzana Tobiásová

Yale UniversityORCID 0000-0003-2750-6215Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Organism:human· 3 Research Focus:cell-cell communication· 2 Approach:experimental assay· 2 Assay:imaging· 2 Assay:scRNA-seq· 2 Approach:statistical model· 2

Frequent Co-Authors

Jordan S. PoberiD· 3 shared Esen SefikiD· 2 shared Richard Anthony FlavelliD· 2 shared Andrew J. MartinsiD· 1 shared Fengrui ZhangiD· 1 shared Gwendolyn K. DavisiD· 1 shared Jennifer Madison McNiffiD· 1 shared John S. TsangiD· 1 shared

Activity by Year

2026-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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3 shared papersImmunology

Jordan S. PoberORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

ImmunologybioRxivPreprintOct 10, 2026

✦ In-depth digest

A surviving beta cell subpopulation enriched in patients with T1D

Maxwell Spurrell, Zuzana Tobiásová, Jordan S. Pober…·Kevan C. Herold

Regulon-based clustering of islet scRNA-seq finds an IRF1-high, cytokine-driven beta cell state with reduced identity and autoantigen genes, enriched in type 1 diabetes donors.

DOI 10.64898/2026.05.15.725449

humanscRNA-seq

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ImmunologyThe Journal of ImmunologyPeer-reviewedJul 28, 2026

Endothelial cell MHC molecules are necessary and sufficient to reject 3D-printed human skin grafts in an advanced human immune system mouse

Zuzana Tobiásová, Esen Sefik, Lingfeng Qin…·Jordan S. Pober

The study shows that endothelial cell MHC molecules are critical for rejecting 3D-printed human skin grafts in an immunocompetent humanized mouse model.

DOI 10.1093/jimmun/vkag141.675

humanmouse

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ImmunologyProceedings of the National Academy of SciencesPeer-reviewedFeb 5, 2026

T Cell Receptors for Antigen on Intraepitheial Cytolytic T Lymphocytes in Celiac Disease Engage Enterocyte HLA-E and HLA-B

Justin E. Johnson, Kriti Agrawal, Rafia S. Al‐Lamki…·Jordan S. Pober

This study identifies IFNγ-producing intraepithelial CTLs in celiac disease that interact with HLA-E/B on enterocytes, promoting CTL recruitment and epithelial killing.

1 citation

humanscRNA-seq

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