Cell BiologybioRxivPreprint
Non-canonical ATR signalling via NBS1 phosphorylation propagates fork slowing from stressed to unperturbed nuclear regions
Jana Krietsch, Ilaria Ceppi, William J. Comstock…·Massimo Lopes
DOI 10.64898/2026.07.09.737522
Author
Alessandro A. Sartori
University of ZurichORCID 0000-0003-2770-0333Unclaimed ORCID profile · follow to get notified of new preprints
Biology Disciplines
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2025-2026 · peak 3/yrCo-Author Collaboration Network
10 top collaborators · 42 mutual co-author linksFocal author (center)Selected co-authorORCID co-authorUnlinked name
Curved chords = mutual co-authorship5 papers
Recent·Most cited
- Cell BiologyEMBO ReportsPeer-reviewed
CIP2A Mediates the Recruitment of the SLX4-MUS81-XPF Tri-Nuclease Complex in Mitosis and Protects Against Replication Stress
Alice Meroni, Annica Pellizzari, Nathalie Varisco…·Manuel Stucki
This paper shows CIP2A recruits the SMX tri-nuclease complex to manage under-replicated DNA during mitosis, preventing chromosome segregation errors in HRD cancers.
3 citationstop 3% - Molecular BiologyNature CommunicationsPeer-reviewed
USP7 deubiquitinase stabilizes FAN1 to support DNA crosslink repair and suppress CAG repeat expansion
Giulio Collotta, Marco Gatti, Irina-Maria Ungureanu…·Alessandro A. Sartori
This study identifies USP7 as a stabilizer of FAN1, showing its role in DNA crosslink repair and suppression of CAG repeat expansion in Huntington's disease.
DOI 10.1038/s41467-026-70051-9 - ImmunologyCell ReportsPeer-reviewed
Cancer-cell-derived cGAMP limits the activity of tumor-associated CD8+ T cells
Michael Herbst, Hakan Köksal, Silvan Brunn…·Maries F. van den Broek
This study shows that cancer-cell-derived cGAMP both enhances and limits CD8+ T cell anti-tumor activity through STING signaling.
7 citations - Molecular BiologyNucleic Acids ResearchPeer-reviewed
The PIN1-p38-CtIP signaling axis protects stalled replication forks from deleterious degradation
Francesca Vivalda, Marco Gatti, Letizia Manfredi…·Alessandro A. Sartori
This study identifies the PIN1-p38-CtIP signaling pathway as essential for protecting stalled replication forks from degradation, independent of its role in homologous recombination.
3 citations