Christopher Albanese

This study shows that anti-Aβ immunotherapy in APOE4 mice increases cerebral amyloid angiopathy and vascular damage, while APOE3 mice show no significant changes.

This study identifies SLC25A1 as a regulator of PD-L1 and type I interferon signaling via a mitochondrial pathway, influencing tumor immune response and immunotherapy efficacy.

This study shows that SLC25A1 deficiency in mice causes senescence via p53-dependent pathways, leading to developmental disorders similar to human D/L-2HGA.