Author

Elisabetta Ciani

University of BolognaORCID 0000-0003-3130-5168Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Assay:electronic health records· 1

Frequent Co-Authors

Giorgio MediciiD· 2 shared Giulia SagonaiD· 2 shared Maurizio GiustettoiD· 2 shared Tommaso PizzorussoiD· 2 shared Alessandra RaspantiiD· 1 shared Alysson Renato MuotriiD· 1 shared Andrea Macioce· 1 shared Anthony BattagliaiD· 1 shared

Activity by Year

2021-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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2 shared papersNeuroscience

Giorgio MediciORCID

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2 papers

Recent·Most cited

NeuroscienceProgress in NeurobiologyPeer-reviewedAug 19, 2026

Postnatal Sensory Experience and Barrel Cortex Alterations Anticipate Autistic Traits in a Mouse Model of Cdkl5 Deficiency Disorder

Alessandra Raspanti, Riccardo Pizzo, Antonia Gurgone…·Maurizio Giustetto

This study shows that CDKL5 deficiency in mice leads to sensory processing defects in the barrel cortex and autistic traits, which can be rescued by neonatal CDKL5 replacement.

1 citation

mouseelectronic health records

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NeurosciencebioRxivPreprintJul 26, 2021

EXPRESSION OF A SECRETABLE, CELL-PENETRATING CDKL5 PROTEIN ENHANCES THE EFFICACY OF AAV VECTOR-MEDIATED GENE THERAPY FOR CDKL5 DEFICIENCY DISORDER.

Giorgio Medici, Marianna Tassinari, Giuseppe Galvani…·Elisabetta Ciani

This study shows that using a secretable TATk-CDKL5 protein in AAV gene therapy improves CDKL5 replacement and functional outcomes in Cdkl5 KO mice compared to native CDKL5 expression.

DOI 10.1101/2021.07.26.453746

mouseprotein sequence

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