Stuart J. D. Neil

This study shows that Gag proteolysis drives HIV-1 Env conformational closure through the gp41 cytoplasmic tail, revealing a mechanism for immune evasion.

This study shows that m6A RNA modifications regulate antiviral gene expression by limiting IFITM2/3 expression during influenza A virus infection.

This study identifies how the long ZAP isoform recruits enzymes to degrade viral RNA through a coordinated decay pathway.

The paper identifies diverse spike haplotypes in persistent SARS-CoV-2 infections that show immune escape mechanisms similar to variants of concern.

Alpha-BET uses AlphaFold to identify optimal sites for labeling viral glycoproteins with nanobodies, enabling high-resolution imaging of viral structures without disrupting their function.

This study characterizes the KHNYN ex-diKH domain's role in ZAP antiviral activity, revealing its structural features and identifying a potential protein-protein interaction site.