Natalia Duque‐Wilckens

The paper investigates the distinct behavioral and electrophysiological roles of oxytocin neurons in the anterior and posterior paraventricular nucleus using genetic and pharmacological manipulations in mice.

This study shows developmental PBDE exposure impairs mast cell function in adult mice by disrupting granule maturation and calcium signaling, leading to reduced histamine release.

This study shows that FosB/ΔFosB in mast cells regulates gene expression to reduce allergic inflammation in male mice by enhancing DUSP4 expression.

This study shows that KDM2B dysfunction leads to neural stem cell senescence and ASD/ID-like behaviors in mice, linking 12q24.31 microdeletions to neurodevelopmental disorders.

This study shows that loss of ASH1L in developing mouse brains leads to autistic-like behaviors and cognitive impairments, establishing a causal link between ASH1L mutations and ASD/ID.

The paper shows that specific extra-hypothalamic oxytocin neurons regulate stress-induced social anxiety behaviors by modulating social vigilance and approach.

This study shows that HDACi treatment promotes caregiving behavior in non-aggressive male mice and changes IEG expression patterns in fear/defensive circuits.