Martin R.J. Kolb

This paper develops an ex vivo model using murine precision-cut lung slices to study profibrotic macrophage polarization and validates its utility for fibrosis research.

This study identifies defective mesenchymal Bmpr1a-mediated BMP signaling as a cause of congenital pulmonary cysts by disrupting lung branching morphogenesis and airway structure.

This study shows that Gremlin, an inhibitor of BMP, promotes AT2 cell proliferation and differentiation into AT1s during lung injury, facilitating alveolar repair.