Jaswinder Singh Maras

This study shows that a high-protein donor diet enhances fecal microbiota transplantation efficacy in alcoholic liver disease by increasing caproic acid and PPARα signaling.

The paper examines temporal changes in sEV cargo (miRNA and proteins) in UV-C-damaged C. elegans to identify molecular signatures associated with damage, response, and recovery phases.

This study identifies a VDR-independent mineralization pathway in osteoblasts induced by γ-linolenic acid through the CaMKII-SMAD2/3 pathway, offering a metabolic strategy to restore bone formation under vitamin D deficiency.

This study identifies dysregulated proteins in serum-derived exosomes from Moyamoya disease patients, linking them to angiogenesis and vascular signaling pathways.

This study profiles the metabolome of Ashwagandha to identify bioactive compounds and their associations with therapeutic functions.

This study shows methionine supplementation enhances pro-inflammatory responses and mycobacterial clearance in tuberculosis by modulating host metabolism and epigenetic pathways.

The paper identifies Streptococcal Scl1 as a bacterial protein that activates TGFβR1 signaling, driving fibrosis-inflammation in alcohol-associated liver disease through gut microbiota alterations.

The paper identifies PS1145 as a potential therapeutic for reducing systemic inflammation in alcohol-related liver disease by inhibiting IKK phosphorylation and TLR dimerization.

This study identifies elevated chenodeoxycholic acid as a poor prognostic marker in acute liver failure and shows that Bacteroides intestinalis-AM1 reduces bile acids and inflammation, improving liver injury.

This study shows that time-restricted feeding improves metabolic health in mice but has limited effect on reducing Mycobacterium tuberculosis burden early after infection.

This study identifies liver-enriched DPP4 as a driver of gut inflammation in diabetes by inhibiting autophagy, leading to increased immune cell infiltration and colitis.

This study identifies a mitochondrial quality control mechanism that reverses phagosome maturation arrest caused by Mycobacterium tuberculosis through RAB7 recruitment and lysosomal targeting.

This study uses multi-omics analysis to show aspirin reduces liver fibrosis in mice by altering proteome, metabolome, and microbiome pathways linked to inflammation and oxidative stress.