Author

Marisol Hooks

University of PennsylvaniaORCID 0000-0003-4155-8775Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:drug response / drug discovery· 2 Research Focus:perturbation response prediction· 2 Assay:CRISPR screen· 1 Organism:human· 1

Frequent Co-Authors

Elizabeth A. HelleriD· 2 shared Julia J. WinteriD· 2 shared Kiara L. Rodríguez-AcevedoiD· 2 shared Kyle S. Czarnecki· 2 shared Aarthi RamakrishnaniD· 1 shared Alexandra Goldhamer· 1 shared Annabel K. SangreeiD· 1 shared Benjamin L. ProsseriD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersNeuroscience

Elizabeth A. HellerORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

GeneticsbioRxivPreprintJun 20, 2026

A novel preclinical mouse model recapitulates progressive phenotypes of Bryant-Li-Bhoj Syndrome

Dana E. Layo-Carris, Emily L. Durham, Emily E. Lubin…·Elizabeth J. K. Bhoj

This paper describes a novel mouse model of Bryant-Li-Bhoj Syndrome that recapitulates key developmental and behavioral phenotypes, providing a platform for therapeutic testing.

DOI 10.64898/2026.06.16.732665

mouseexperimental assay

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NeurosciencebioRxivPreprintNov 20, 2025

Epigenetic editing of Cartpt promotes acquisition and extinction of cocaine memory

Julia J. Winter, Marisol Hooks, Keegan S. Krick…·Elizabeth A. Heller

The paper uses CRISPR-based epigenetic editing to manipulate Cartpt expression in mice, showing it affects cocaine memory acquisition and extinction.

3 citations

mouseexperimental assay

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NeurosciencebioRxivPreprintOct 28, 2025

CRISPR-mediated transcriptional activation as a mutation-independent therapeutic strategy for SYNGAP1-related intellectual disability

Laura Sichlinger, Molly B. Reilly, Sakshi Arora…·Elizabeth A. Heller

This paper demonstrates that CRISPR-mediated transcriptional activation can upregulate SYNGAP1 in a mouse model of SRID and human neurons, rescuing deficits without targeting mutations.

3 citations

mousehuman

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